Question # 1 (Multiple
Choice) Relapse rate for do although ulcer following monotherapy treatment with
H2 receptor blockers:
A) 95 %
B) 75%
C) 50%
D) 15%
E) 5%
Question # 2 (Multiple Answer) Physiological stimulation gastric acid secretion -- phases associated with food intake:
A) cephalic phase
B) gastric phase
C) intestinal phase
Question # 3 (Multiple Answer) Physiological/pathophysiological effects of Helicobacter pylori:
A) proinflammatory
B) degradation of glycoprotein-lipid mucus layer complexes
C) production of damaging bacterial proteins
D) causes active, chronic gastritis
Question # 4 (Multiple Choice) Acid secretion phase, following food intake, defined by stimulation of mechanical and chemical gastric wall receptors by luminal contents:
A) cephalic phase
B) gastric phase
C) intestinal phase
Question # 5 (Multiple Choice) Mechanism(s) by which somatostatin reduces gastrin release:
A) inhibits parietal cells accretion
B) inhibits histamine release by enterochromaffin-like cells
C) both
D) neither
Question # 6 (Multiple Choice) Positive factor: Zollinger-Ellison syndrome --
A) Helicobacter pylori
B) gastrin-secreting islet cell tumor
C) both
D) neither
Question # 7 (Multiple Choice) Pathogenic factor(s) in duodenal ulcer:
A) COPD (chronic obstructive pulmonary disease)
B) genetic factor
C) cigarette smoking
D) alcoholic cirrhosis
E) all of the above
Question # 8 (Multiple Answer) Example(s) of "protective factor(s)" in peptic ulcer disease:
A) gastric mucus
B) prostaglandins
C) pepsins
D) bicarbonate
E) Helicobacter pylori
Question # 9 (True/False) There exists a direct correlation between pepsinogen I serum concentrations and maximal gastric acid secretion:
A) true
B) false
Question # 10 (Multiple Choice) Peptic ulcer classification:
A) duodenal
B) gastric
C) both
D) neither
Question # 11 (Multiple Answer) Characteristic(s) of gastric mucosal acid secretion:
A) oxidative dephosphorylation dependent
B) from parietal cells founded in mucosal glands of fundusof the stomach
C) stimulated by muscarinic cholinergic system (parietal cell innervation)
D) most potent stimulant -- histamine direct action
Question # 12 (Multiple Answer) Activation of these/this receptor(s) on basolateral parietal cells inhibit(s) gastric acid secretion
A) histamine
B) gastrin
C) prostaglandins
D) acetylcholine
Question # 13 (Multiple Choice) Relapse rate for duodenalulcer following H. pylori eradication:
A) 90%
B) 75%
C) 50%
D) 15%
E) 5%
Question # 14 (Multiple Answer) Histamine and gastric acid secretion:
A) released from enterochromaffin-like cells
B) release enhanced by increased cholinergic activity
C) most important gastric acid secretion stimulant
Question # 15 (Multiple Answer) Inhibition of basal acid secretion:
A) cimetidine (Tagamet)
B) histamine
C) nizatidine (Axid)
D) ranitidine (Zantac)
E) famotidine (Pepcid)
Question # 16 (Multiple Choice) Location(s) of histamine in gastric mucosa:
A) enterochromaffin-like cells (ECL)
B) mast cell cytoplasmic granules
C) both
D) neither
Question # 17 (Multiple Choice) Ranitidine (Zantac) and gastric acid secretion:
A) inhibits basal acid secretion
B) inhibits secretion in response to vagal stimulation or feeding
C) both
D) neither
Question # 18 (Multiple Choice) Example(s) of "aggressive factors" in peptic ulcer disease:
A) gastric acid
B) pepsin
C) both
D) neither
Question # 19 (Multiple Answer) Increased incidence of duodenal ulcer associated with:
A) chronic renal failure
B) alcoholic cirrhosis
C) renal transplantation
D) systemic mastocytosis
E) hyperparathyroidism
Question # 20 (Multiple Answer) Basolateral parietal cell membranes contained these receptor types:
A) gastrin
B) acetylcholine
C) prostaglandins
D) histamine
Correct Answers
Question # 1 (Multiple Choice) Relapse rate for do although ulcer following monotherapy treatment with H2 receptor blockers:
Answer: (B) 75%
Question # 2 (Multiple Answer) Physiological stimulation gastric acid secretion -- phases associated with food intake:
(A) cephalic phase
(B) gastric phase
(C) intestinal phase
Question # 3 (Multiple Answer) Physiological/pathophysiological effects of Helicobacter pylori:
(A) proinflammatory
(B) degradation of glycoprotein-lipid mucus layer complexes
(C) production of damaging bacterial proteins
(D) causes active, chronic gastritis
Question # 4 (Multiple Choice) Acid secretion phase, following food intake, defined by stimulation of mechanical and chemical gastric wall receptors by luminal contents:
Answer: (B) gastric phase
Question # 5 (Multiple Choice) Mechanism(s) by which somatostatin reduces gastrin release:
Answer: (C) both
Question # 6 (Multiple Choice) Positive factor: Zollinger-Ellison syndrome --
Answer: (C) both
Question # 7 (Multiple Choice) Pathogenic factor(s) in duodenal ulcer:
Answer: (E) all of the above
Question # 8 (Multiple Answer) Example(s) of "protective factor(s)" in peptic ulcer disease:
(A) gastric mucus
(B) prostaglandins
(D) bicarbonate
Question # 9 (True/False) There exists a direct correlation between pepsinogen I serum concentrations and maximal gastric acid secretion:
Answer: True
Question # 10 (Multiple Choice) Peptic ulcer classification:
Answer: (C) both
Question # 11 (Multiple Answer) Characteristic(s) of gastric mucosal acid secretion:
(A) oxidative dephosphorylation dependent
(B) from parietal cells founded in mucosal glands of fundusof the stomach
Question # 1 (Multiple Choice) Relapse rate for do although ulcer following monotherapy treatment with H2 receptor blockers:
Answer: (B) 75%
Question # 2 (Multiple Answer) Physiological stimulation gastric acid secretion -- phases associated with food intake:
(A) cephalic phase
(B) gastric phase
(C) intestinal phase
Question # 3 (Multiple Answer) Physiological/pathophysiological effects of Helicobacter pylori:
(A) proinflammatory
(B) degradation of glycoprotein-lipid mucus layer complexes
(C) production of damaging bacterial proteins
(D) causes active, chronic gastritis
Question # 4 (Multiple Choice) Acid secretion phase, following food intake, defined by stimulation of mechanical and chemical gastric wall receptors by luminal contents:
Answer: (B) gastric phase
Question # 5 (Multiple Choice) Mechanism(s) by which somatostatin reduces gastrin release:
Answer: (C) both
Question # 6 (Multiple Choice) Positive factor: Zollinger-Ellison syndrome --
Answer: (C) both
Question # 7 (Multiple Choice) Pathogenic factor(s) in duodenal ulcer:
Answer: (E) all of the above
Question # 8 (Multiple Answer) Example(s) of "protective factor(s)" in peptic ulcer disease:
(A) gastric mucus
(B) prostaglandins
(D) bicarbonate
Question # 9 (True/False) There exists a direct correlation between pepsinogen I serum concentrations and maximal gastric acid secretion:
Answer: True
Question # 10 (Multiple Choice) Peptic ulcer classification:
Answer: (C) both
Question # 11 (Multiple Answer) Characteristic(s) of gastric mucosal acid secretion:
(A) oxidative dephosphorylation dependent
(B) from parietal cells founded in mucosal glands of fundusof the stomach
(C) stimulated by
muscarinic cholinergic system (parietal cell innervation)
most potent: gastrin
Question # 12 (Multiple Answer) Activation of these/this receptor(s) on basolateral parietal cells inhibit(s) gastric acid secretion
(C) prostaglandins
Question # 13 (Multiple Choice) Relapse rate for duodenalulcer following H. pylori eradication:
Answer: (D) 15%
Question # 14 (Multiple Answer) Histamine and gastric acid secretion:
(A) released from enterochromaffin-like cells
(B) release enhanced by increased cholinergic activity
(C) most important gastric acid secretion stimulant
Question # 15 (Multiple Answer) Inhibition of basal acid secretion:
(A) cimetidine (Tagamet)
(C) nizatidine (Axid)
(D) ranitidine (Zantac)
(E) famotidine (Pepcid)
Question # 16 (Multiple Choice) Location(s) of histamine in gastric mucosa:
Answer: (C) both
Question # 17 (Multiple Choice) Ranitidine (Zantac) and gastric acid secretion:
Answer: (C) both
Question # 18 (Multiple Choice) Example(s) of "aggressive factors" in peptic ulcer disease:
Answer: (C) both
Question # 19 (Multiple Answer) Increased incidence of duodenal ulcer associated with:
(A) chronic renal failure
(B) alcoholic cirrhosis
(C) renal transplantation
(D) systemic mastocytosis
(E) hyperparathyroidism
Question # 20 (Multiple Answer) Basolateral parietal cell membranes contained these receptor types:
(A) gastrin
(B) acetylcholine
(C) prostaglandins
(D) histamine
most potent: gastrin
Question # 12 (Multiple Answer) Activation of these/this receptor(s) on basolateral parietal cells inhibit(s) gastric acid secretion
(C) prostaglandins
Question # 13 (Multiple Choice) Relapse rate for duodenalulcer following H. pylori eradication:
Answer: (D) 15%
Question # 14 (Multiple Answer) Histamine and gastric acid secretion:
(A) released from enterochromaffin-like cells
(B) release enhanced by increased cholinergic activity
(C) most important gastric acid secretion stimulant
Question # 15 (Multiple Answer) Inhibition of basal acid secretion:
(A) cimetidine (Tagamet)
(C) nizatidine (Axid)
(D) ranitidine (Zantac)
(E) famotidine (Pepcid)
Question # 16 (Multiple Choice) Location(s) of histamine in gastric mucosa:
Answer: (C) both
Question # 17 (Multiple Choice) Ranitidine (Zantac) and gastric acid secretion:
Answer: (C) both
Question # 18 (Multiple Choice) Example(s) of "aggressive factors" in peptic ulcer disease:
Answer: (C) both
Question # 19 (Multiple Answer) Increased incidence of duodenal ulcer associated with:
(A) chronic renal failure
(B) alcoholic cirrhosis
(C) renal transplantation
(D) systemic mastocytosis
(E) hyperparathyroidism
Question # 20 (Multiple Answer) Basolateral parietal cell membranes contained these receptor types:
(A) gastrin
(B) acetylcholine
(C) prostaglandins
(D) histamine
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